PATIENT JOURNEY

Steroids, Stress, Sugar: The Loop Nobody Had Drawn for Me

Published September 2026 © 2026 Chin Qi Yong ~5 min read

For years I treated the patch. Nobody had drawn me the circle it sits inside — the cream, the stress, the food, and the blood sugar quietly linking all three.

For years, every appointment about my skin had the same shape. I showed the patch. Someone looked at the patch. I left with something to put on the patch. It worked, mostly — the way bailing water works on a boat that is still taking it in.

Then I sat down with a doctor who did something nobody had done in all those years. He stopped talking about the patch. He drew a circle instead, and started filling in what feeds it.

I had been managing one corner of a loop and calling it the whole problem.

What is the cream actually doing once it is in?

The cream calms things down. That is its job, it does it well, and I have no interest in talking anyone out of it. But it does not stay politely on the surface the way I had always pictured. A little of it goes through, and how much depends on where you put it, how much skin you cover, how long you keep going — and, the part I never knew, on how angry the skin already is.

Broken, scaling, inflamed skin lets more through than calm skin does. Not a little more, either. So the worse a stretch gets, the more of it I was applying, over more of me, into skin that was more open than usual. The flare quietly widens its own door.

For most people this stays academic — used on the area and for the stretch a doctor sets, the wider effects are uncommon, and the medical sources are clear about that. It stops being academic when you have been holding a large area for years, which is where I had ended up without ever deciding to. That arithmetic is exactly why the dose and the duration belong to the doctor and not to me, or to anyone on the internet.

Why does stress belong in the same sentence as a steroid?

Here is the bit that reorganised my thinking, and it is almost embarrassingly simple once someone says it out loud.

Cortisol — the thing your body pours out on the week everything lands at once — is the body's own version of the same class of medicine that is in the tube. They are relatives. That is why the medicine works: it is standing in for something you already make.

So it is worth asking what your own version does when it shows up. Among other jobs, it goes to the liver and tells it to put sugar into the blood. That is the design working exactly as intended — it is fuel, released for a fight or a run. In my case the emergency was a spreadsheet, a client, and a decision I had been turning over since Tuesday. No running involved. The fuel gets released anyway, and then just sits there.

Which means the stressful month and the tube in the bathroom were leaning on the same lever, from two different directions, without either one telling me.

So where does the sugar come in?

Then the third input, the one I could not blame on anything but myself: what I eat, and when. Late dinners because the day ran long. The teh tarik that is really a small dessert. Eating at ten because I forgot to at seven.

Three taps, one basin. And the basin was rarely getting a chance to drain.

What made me sit back was learning the traffic runs both ways. Skin conditions like mine and blood-sugar trouble turn up together far more often than chance would explain — that has been measured across very large groups of people, not guessed at. And the more stubborn the skin tends to be, the more the sugar picture tends to shift with it. A body that is handling sugar badly is a body running warm on the inside. That warmth is not separate from what is happening on my elbows. It is the same fire, seen from a different room.

So: sugar feeds the inflammation, the inflammation feeds the flare, the flare gets more cream and more worry, and the worry and the cream both feed the sugar. Round it goes. Nobody in that circle is the villain. The circle is the problem.

What actually changed after that appointment?

First, what did not change: my medication. I did not reduce it, skip it, taper it, or take the break the internet is always so eager to recommend. That call sits with the person who can examine me and read my results, and handing it to a stranger — or to myself at 1am — is how people get hurt. If the loop I have described sounds like yours, that is a conversation to have with your doctor or pharmacist, not a cue to change anything on your own.

What changed were the inputs I actually own. Sleep, first, because it is the cheapest one and I had been treating it as optional. Food, second — less about virtue than about timing and not eating my largest meal closest to bed. Stress, third, which is the hardest and the one I am still bad at.

And measurement. I got the blood work done, which I had avoided for years in the way you avoid checking a bank balance. You cannot manage a loop you refuse to look at.

The change that mattered most was not a habit at all. It was that I stopped thinking about my skin as a skin problem. It is a whole-body situation that happens to show up where everyone can see it.

If you have been fighting the same patch for years, the most useful question may not be what else to put on it — but what else is feeding it. That is a question for someone who can actually measure you.

This article is general skin information, not medical advice. It is one person's experience and it is not a guide to diagnosing anyone, changing any prescription, or choosing what to buy or use. Decisions about medication — including how much, how long, and when to stop — belong to your doctor or pharmacist. If your skin keeps flaring and not settling, that is worth raising with them. Last reviewed: September 2026.

Sources

  1. StatPearls (NCBI Bookshelf) — “Topical Corticosteroids.” Gabros, Nessel & Zito, updated 26 April 2025. Absorption is influenced by formulation, the state of the skin barrier and occlusion; penetration “increases 2- to 10-fold in diseased states, such as inflammation and desquamation.” Systemic effects “are less likely due to low percutaneous absorption” but can develop with prolonged use of high-potency steroids, and include HPA-axis suppression, Cushing syndrome, hypertension and hyperglycaemia. Low-to-medium potency is advised for large surface areas; treatment duration is generally not more than 2 to 4 weeks regardless of potency, under a doctor's supervision.
  2. Kuo, Harris & Wang — Molecular and Cellular Endocrinology — “Regulation of Glucose Homeostasis by Glucocorticoids.” Glucocorticoids promote gluconeogenesis in the liver, while in skeletal muscle and white adipose tissue they reduce glucose uptake and utilisation by opposing the action of insulin.
  3. Practical guidance, Therapeutic Advances in Endocrinology and Metabolism — “Practical Guide to Glucocorticoid Induced Hyperglycaemia and Diabetes.” Glucocorticoids contribute to raised blood glucose both by increasing insulin resistance and by decreasing insulin secretion.
  4. Meta-analysis — “Psoriasis and Metabolic Disorders: A Comprehensive Meta-Analysis of Million Adults Worldwide.” Pooled analysis across very large populations reports a higher prevalence of metabolic disorders, including disturbed glucose handling, among people with psoriasis than in comparison groups.
  5. György & colleagues — “Psoriasis Severity — A Risk Factor of Insulin Resistance Independent of Metabolic Syndrome.” Reports a correlation between psoriasis severity and measured insulin resistance, with severity behaving as a risk factor independent of metabolic syndrome. Figures vary between studies and populations.
  6. Review — “Insulin Resistance, Metabolic Syndrome, and Inflammatory Skin Disease.” Describes the relationship as bidirectional: insulin resistance can worsen inflammation and oxidative stress, which in turn can worsen metabolic control.

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